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Új cikk:
Ragó A, Honbolygó F, Róna Z, Beke A, Csépe V. (2014) Effect of maturation on suprasegmental speech processing in full- and preterm infants: A mismatch negativity study. Research in Developmental Disabilities. Vol 35 (1), 192–202.

Abstract:
Infants born prematurely are at higher risk for later linguistic deficits present in delayed or atypical processing of phonetic and prosodic information. In order to be able to specify the nature of this atypical development, it is important to investigate the role of early experience in language perception. According to the concept of Gonzalez-Gomez and Nazzi (2012) there is a special intrauterine sensitivity to the prosodic features of languages that should have a special role in language acquisition. Therefore, we may also assume that pre- and full-term infants having months difference in intrauterine experience show different maturation patterns of processing prosodic and phonetic information present at word level. The aim of our study was to investigate the effect of these differences on word stress pattern vs. phoneme information processing.
Two age groups of infants (6 and 10 month-olds) were included in our study. 21 of 46 of the total of infants investigated were prematurely born with low birth weight.
We used the mismatch negativity (MMN) event related brain potential (ERP) component, a widely used electrophysiological correlate of acoustic change detection, for testing the assumed developmental changes of phoneme and word stress discrimination. In a passive oddball paradigm we used a word as standard, a pseudo- word as phoneme deviant, and an illegally uttered word as stress deviant.
Our results showed no differences in MMN responses in the phoneme deviant condition between the groups, meaning a relatively intact maturation of phoneme processing of preterm infants as compared to their contemporaries. However, the mismatch responses measured in the stress condition revealed significant between-group differences. These results strengthen the view that the total length of intrauterine experience influences the time of emergence of prosodic processing.

Kóbor, A., Takács, Á., Honbolygó, F., Csépe, V. (2014) Generalized lapse of responding in trait impulsivity indicated by ERPs: the role of energetic factors in inhibitory control. International Journal of Psychophysiology, 92:(1), 16-25.

Impaired inhibitory control is one of the still debated underlying mechanisms of trait impulsivity. The Cognitive Energetic Model accounts for the role of energetic factors mediating task performance. The aim of the present study was to compare inhibitory control functions of adults with high and low impulsivity by using a modified Eriksen flanker task. Adults were classified as impulsive (n = 15) and control (n = 15) participants based on the Barratt Impulsiveness Scale. Flanker trials had three levels of required effort manipulated by visual degradation. We analyzed RT, accuracy, and ERPs time-locked to the flanker stimuli. Reaction time of impulsive participants was generally slower than that of controls', but accuracy was similar across groups. N2c showed that monitoring of response conflict was modulated by task requirements independent of impulsivity. The P3 latency was delayed in the impulsive group indicating slower stimulus evaluation. The P3 amplitude was reduced in the control group for moderately degraded incongruent trials suggesting that the attentional resources were employed less. The Lateralized Readiness Potential (LRP) peaked later in the impulsive group irrespective of experimental effects. The amplitude of the positive-going LRP recorded in the incongruent condition was comparable across groups, but the latency was delayed partly supporting a stronger susceptibility to stimulus interference of impulsive participants. Their delayed incongruent negative-going LRP reflected a weaker response inhibition and a slower correct response organization. In conclusion, impaired inhibitory functions in impulsivity could not be unequivocally demonstrated, but we found a generalized lapse of motor activation.